STAT Stitch Deep Dive Podcast Beyond The Bedside

45:46

CC | Burns

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/13

Burn Classification & TBSA Assessment Burn severity depends on depth, extent (TBSA), location, and patient risk factors. Depth is classified as partial-thickness (epidermal/dermal; painful, red, blistered) or full-thickness (dermal destruction, painless, leathery eschar, visible thrombosed vessels). TBSA is estimated using the Rule of Nines for adults or the Lund-Browder chart for children. Referral to a burn center is indicated for partial-thickness burns >10% TBSA, chemical/electrical

en

22:50

CC | PRMIER Chronic Neurological Complications

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/13

Primary HeadachesTension (TTH): Bilateral, dull bandlike pressure. No nausea. Tx: NSAIDs. Prev: Amitriptyline.Migraine: Unilateral throbbing, ± aura. Triggers: stress, red wine. Acute: Triptans (sumatriptan; contraindicated in CAD, ischemic stroke, uncontrolled HTN). Prev: Topiramate, β-blockers, Botox.Cluster: Severe unilateral orbital pain in clusters. ptosis, tearing. Tx: High-flow 100% O2 (7-12 L/min), triptans. Prev: Verapamil.Medication Overuse (MOH): Daily headache from analgesic overuse. Tx:

en

58:48

CC | Chronic Neurological Complications

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/13

Primary HeadachesTension (TTH): Bilateral, dull bandlike pressure. No nausea. Tx: NSAIDs. Prev: Amitriptyline.Migraine: Unilateral throbbing, ± aura. Triggers: stress, red wine. Acute: Triptans (sumatriptan; contraindicated in CAD, ischemic stroke, uncontrolled HTN). Prev: Topiramate, β-blockers, Botox.Cluster: Severe unilateral orbital pain in clusters. ptosis, tearing. Tx: High-flow 100% O2 (7-12 L/min), triptans. Prev: Verapamil.Medication Overuse (MOH): Daily headache from analgesic overuse. Tx:

en

26:33

CC | PRIMER Stoke [CVA ]

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

Core Concepts & Emergency Recognition A stroke is an emergency where cell death occurs due to ischemia (inadequate blood flow, 87% of cases) or hemorrhage (bleeding, 13%). Interruption of blood flow alters brain metabolism in 30 seconds, stops it in 2 minutes, and causes cell death in 5 minutes. The FAST protocol is critical for rapid recognition: Face drooping, Arm weakness/drift, Speech difficulties, and Time of symptom onset (critical for determining treatment eligibility).Pathophysiology

en

63:15

CC | Stroke [CVA]

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

Core Concepts & Emergency Recognition A stroke is an emergency where cell death occurs due to ischemia (inadequate blood flow, 87% of cases) or hemorrhage (bleeding, 13%). Interruption of blood flow alters brain metabolism in 30 seconds, stops it in 2 minutes, and causes cell death in 5 minutes. The FAST protocol is critical for rapid recognition: Face drooping, Arm weakness/drift, Speech difficulties, and Time of symptom onset (critical for determining treatment eligibility).Pathophysiology

en

25:15

CC | PRIMER ICP

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

Intracranial Regulation & Monro-Kellie Doctrine The skull contains brain tissue (78%), blood (12%), and CSF (10%). Under Monro-Kellie, because this volume is constant, an increase in any one component must be offset by displacing another; otherwise, intracranial pressure (ICP) rises. Compensations include displacing CSF, collapsing cerebral veins, and compressing tissue. Decompensation causes ischemia. Normal ICP is 5 to 15 mm Hg; sustained pressure >20 mm Hg is abnormal.Cerebral Perfusion

en

63:08

CC | ICP

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

Intracranial Regulation & Monro-Kellie Doctrine The skull contains brain tissue (78%), blood (12%), and CSF (10%). Under Monro-Kellie, because this volume is constant, an increase in any one component must be offset by displacing another; otherwise, intracranial pressure (ICP) rises. Compensations include displacing CSF, collapsing cerebral veins, and compressing tissue. Decompensation causes ischemia. Normal ICP is 5 to 15 mm Hg; sustained pressure >20 mm Hg is abnormal.Cerebral Perfusion

en

21:35

CC | PRIMER Neuro Assessment

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. Structure & Cellular BiologyOrganization: The CNS consists of the brain, spinal cord, and CN I-II. The PNS contains CN III-XII, spinal nerves, and the ANS.Cells & Myelin: Neurons conduct action potentials via saltatory conduction across nodes of Ranvier, accelerated by myelin. Astrocytes form the blood-brain barrier and create scar tissue (gliosis) upon injury. Oligodendrocytes myelinate CNS axons; Schwann cells myelinate PNS axons.Synapses: Neurotransmitters cross synapses to alter

en

48:31

CC | Neuro Assessment

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. Structure & Cellular BiologyOrganization: The CNS consists of the brain, spinal cord, and CN I-II. The PNS contains CN III-XII, spinal nerves, and the ANS.Cells & Myelin: Neurons conduct action potentials via saltatory conduction across nodes of Ranvier, accelerated by myelin. Astrocytes form the blood-brain barrier and create scar tissue (gliosis) upon injury. Oligodendrocytes myelinate CNS axons; Schwann cells myelinate PNS axons.Synapses: Neurotransmitters cross synapses to alter

en

17:01

CC | PRIMER CKD & AKI

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. PathophysiologyAKI: Sudden, potentially reversible decline in GFR.Prerenal: Decreased perfusion (hypovolemia, shock, HF). Oliguria occurs without tissue damage; reversible with fluids.Intrarenal: Direct tissue damage, 90% from Acute Tubular Necrosis (ATN) (ischemia, sepsis, nephrotoxins).Postrenal: Mechanical obstruction (BPH, calculi) causing urine reflux.CKD: Gradual, irreversible nephron loss (GFR <60 for >3 months). Primary causes: Diabetes (50%), Hypertension (25%). Stage 5 (ESRD) GFR

en

56:55

CC | CKD & AKI (GU)

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. PathophysiologyAKI: Sudden, potentially reversible decline in GFR.Prerenal: Decreased perfusion (hypovolemia, shock, HF). Oliguria occurs without tissue damage; reversible with fluids.Intrarenal: Direct tissue damage, 90% from Acute Tubular Necrosis (ATN) (ischemia, sepsis, nephrotoxins).Postrenal: Mechanical obstruction (BPH, calculi) causing urine reflux.CKD: Gradual, irreversible nephron loss (GFR <60 for >3 months). Primary causes: Diabetes (50%), Hypertension (25%). Stage 5 (ESRD) GFR

en

29:15

CC | Blood Disorders

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

Core Clinical Concepts Blood disorders disrupt homeostasis. Key pathological threads include:Fatigue: Secondary to tissue hypoxia (anemia, cancer).Infection Risk: Compromised immunity and risk for infection is a major concern in hematologic cancers and a common side effect of therapy.Pain: Common and classic in sickle cell disease (SCD) and oncology.Perfusion & Clotting: Excessive clotting impairs perfusion; inadequate clotting causes blood loss and fluid volume deficit.Anemia: Definition &

en

54:30

CC | GU

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. Anatomy & Blood Supply The upper urinary tract has two retroperitoneal, bean-shaped kidneys (T12–L3) and 2 ureters. The lower tract comprises the bladder and urethra. Each kidney is shielded by a fibrous capsule and cushioned by fat. The functional unit is the nephron (~1 million), consisting of a glomerulus, Bowman's capsule, and tubule system (PCT, loop of Henle, DCT, and collecting ducts). Blood flow is ~1200 mL/min (20-25% of cardiac output) via the renal artery, which divides into

en

31:24

CC | PRIMER Sepsis, SIRS, MODS, and Shock

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. The Continuum: Perfusion FailuresShock: Decreased tissue perfusion causing a life-threatening cellular O2 supply/demand imbalance.SIRS: Systemic inflammation from infection, trauma, or ischemia, causing capillary leak and endothelial damage.Sepsis & Septic Shock: Sepsis is infection + dysregulated host response + organ dysfunction (altered mentation, SBP ≤100, RR ≥22, lactate >1 mmol/L). Septic shock is a subset with persistent hypotension requiring vasopressors to maintain MAP ≥65 mmHg

en

54:23

CC | Sepsis, SIRS, MODS, Shock

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

1. The Continuum: Perfusion FailuresShock: Decreased tissue perfusion causing a life-threatening cellular O2 supply/demand imbalance.SIRS: Systemic inflammation from infection, trauma, or ischemia, causing capillary leak and endothelial damage.Sepsis & Septic Shock: Sepsis is infection + dysregulated host response + organ dysfunction (altered mentation, SBP ≤100, RR ≥22, lactate >1 mmol/L). Septic shock is a subset with persistent hypotension requiring vasopressors to maintain MAP ≥65 mmHg

en

43:28

CC | Dysrhythmia

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

ell Properties & Conduction** Heart cells have four properties: automaticity, excitability, conductivity, contractility [1, 2]. Conduction travels from the SA node (60–100 bpm) through the AV node (40–60 bpm), bundle of His, bundle branches, to Purkinje fibers (20–40 bpm) [1, 3]. Vagal tone slows heart rate (HR); sympathetic activity increases HR and contractility [1]. Resting potential features high internal K+, external Na+; depolarization (Phase 0) is rapid Na+ influx [1, 4]. ECG Basics

en

71:08

CC | Dysrhythmia

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/12

Cell Properties & Conduction** Heart cells have four properties: automaticity, excitability, conductivity, contractility [1, 2]. Conduction travels from the SA node (60–100 bpm) through the AV node (40–60 bpm), bundle of His, bundle branches, to Purkinje fibers (20–40 bpm) [1, 3]. Vagal tone slows heart rate (HR); sympathetic activity increases HR and contractility [1]. Resting potential features high internal K+, external Na+; depolarization (Phase 0) is rapid Na+ influx [1, 4]. ECG Basics

en

19:04

POD | Welcome to the Podcast

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/11

Season 1 = Health Assessment Season 2 = Med\Surg Season 3 = Pharmacology Season 4 = Pediatrics Season 5 = Mom\ BabySeason 6 = Mental Health Season 7 = Critical Care Season 19 = ACLS Certification Season 20 = PALS Certification Season 30 = ATLS Certification (Trauma Certification for MDs)

en

21:43

CC | PRIMER CAD/ ACS

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/11

Atherosclerosis is the foundational pathology of coronary artery disease (CAD). It is a progressive inflammatory disease characterized by lipid deposition within the intima of the arterial wall, triggered by chronic endothelial injury from factors like tobacco smoking, hypertension, hyperlipidemia, and diabetes. The disease progresses through three key developmental stages over several decades:Fatty Streak: The earliest lesions, consisting of lipid-filled smooth muscle cells that appear yellow,

en

23:00

CC | PRIMER Heart Failure

STAT Stitch Deep Dive Podcast Beyond The Bedside ·

2026/08/11

1. Core Typology & Risk FactorsHFrEF (Systolic): EF < 40%. LV is dilated/hypertrophied, unable to eject blood effectively, reducing CO.HFpEF (Diastolic): EF ≥ 50%. LV is stiff, noncompliant, causing high filling pressures and decreased filling volume.Causes: HTN and CAD are primary. Long-term HTN control reduces HF incidence by 50%.2. Pathophysiology: The Compensation TrapTrigger: Myocardial injury -> decreased CO.Cascade:RAAS: Renal hypoperfusion triggers renin release, converting

en

All Episodes

Similar Podcasts

Edutainment Learning

Ralph Krauss

Daily Facts

Amalia Dupray and Montgomery Jones.

Les dents et dodo

BFMTV

Education Futures

Svenia Busson & Laurent Jolie

Out In The Hall

NHSA